The process by which alcohol liver disease develops is called alcohol-induced hepatic encephalopathy (AAH). A fatty liver is a condition where there is an overabundance of the liver’s tissues, specifically the cells that make up the liver. Alcohol can cause a considerable amount of damage to these cells.
Diagnosis of alcohol liver disease is based largely on the severity of the disorder. Medical testing is usually required to determine the severity and type of alcohol-induced hepatic steatosis. Treatment of alcohol liver disease also generally depends on the severity of the disorder. Medical treatment usually involves reduction of alcohol consumption. Nutrition support as well as weight loss are also common treatment methods. Screening for fatty liver with ultrasound every six months or ultrasound screening for esophageal cancer with magnetic resonance imaging (MRA) in patients with cirrhosis and serious fatty liver, either with or without obstruction are other methods used to determine the progression of the disease.
Alcoholic hepatitis is a less severe form of alcohol liver disease. It does not result from heavy alcohol use and does not cause cirrhosis or death. A variety of inflammation causes can contribute to alcoholic hepatitis. Alcohol intake is one of them. The condition is characterized by inflammation of the liver cells, which causes acute and chronic liver damage.
Acute alcohol liver disease is characterized by inflammation of liver cells, usually resulting from alcohol abuse. Chronic alcohol abuse is characterized by chronic inflammation of liver cells. Both conditions are associated with development of cirrhosis, but fatty liver does not normally lead to cirrhosis if there is no alcohol abuse. Hepatitis also occurs in alcohol abusers, and it can cause temporary loss of consciousness and other symptoms such as fever, chills, swollen lymph nodes, and vomiting.
There are two forms of alcoholic hepatitis: acute and chronic. Acute alcohol hepatitis is caused by damage to some of the body’s tissues that produce enzymes necessary for the manufacture of bile acids, and the disease is therefore called liver inflammation. On the other hand, chronic alcoholic hepatitis is caused by damage to certain specific organs, which affect the functioning of the body. In this condition, the damage is often extensive and irreversible. This condition is more likely to result in cirrhosis than in acute hepatitis.
Portal hypertension is a liver disease in which excess fluid builds up in the abdominal cavity (ascites), preventing the normal drainage of wastes. The condition develops when the portal vein enlarges due to fluid accumulation, obstructing the drainage of the liver. This causes the accumulation of fluid in the abdomen (ascites) that becomes a cause of discomfort and sometimes pain.
Hepatitis and alcoholic liver disease often appear together because both conditions involve the bacteria that make up the human immune system. Some forms of hepatitis B are associated with autoimmune disorders such as Crohn’s disease or rheumatoid arthritis, while others are genetic. There are certain types of hepatitis C that can be caused by genetic differences and environmental factors. A recent development in medical research is hepatitis D, whereby certain people are more genetically prone to the disease than others.
Alcohol liver disease can be prevented, but only if the primary causes of the problem are addressed. The primary causes are excessive alcohol consumption, the presence of other infections within the body, and damage to certain organ systems. This damage to the liver is either directly or indirectly caused by the excessive alcohol consumption. It may either be caused by the excess alcohol intake or by the infection of the liver cells. To prevent further damage to the liver, alcohol liver disease prevention should be combined with proper diet and nutrition, regular exercise, and avoidance of infections to the liver cells.